Dysmenorrhea
Basics
Description
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Pelvic pain related to menses
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Primary dysmenorrhea: pain without pelvic pathology; diagnosis of exclusion
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Secondary dysmenorrhea: due to underlying pelvic pathology, often more severe and treatment-resistant
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Severity:
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Mild: rarely limits function, seldom needs analgesics
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Moderate: affects daily activity, rare absenteeism
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Severe: daily impairment, absenteeism, poor response to treatment
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Epidemiology
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Primary: starts 6β12 months post-menarche, common in teens to 20s
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Secondary: more often in 20sβ30s
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Up to 90% of menstruating women have primary dysmenorrhea
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Up to 42% miss school/work due to symptoms
Etiology & Pathophysiology
Primary:
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Increased PGF2Ξ± from progesterone drop β hypercontractility, vasoconstriction, uterine ischemia
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C nerve fiber sensitization causes pain
Secondary:
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Endometriosis (most common)
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Adenomyosis
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Congenital uterine anomalies
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PID, fibroids, polyps, ovarian cysts
Risk Factors
Primary:
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Smoking, alcohol
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Early menarche (<12 years)
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Family history
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Heavy/irregular flow
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Nulliparity, non-use of OCPs
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Psychological factors
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History of sexual abuse
Secondary:
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Pelvic infection
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Recent IUD use
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Family history of endometriosis
Prevention
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Primary: exercise, hormonal contraception, childbirth
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Secondary: STI prevention
Associated Conditions
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Heavy/irregular bleeding
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Anxiety, depression
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Lowered QoL
Diagnosis
History
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Primary: cyclic cramping starting at or before menses
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Associated: nausea, diarrhea, headache, fatigue, back/thigh pain
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Relief with NSAIDs, heat, orgasm
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Secondary:
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Pain between cycles, dyspareunia, chronic pelvic pain
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Onset >25 years, progressive severity, poor NSAID response
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Physical Exam
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Primary: normal
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Secondary: findings of infection, uterine abnormalities
Differential Diagnosis
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Primary: clinical history
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Secondary: endometriosis, PID, ectopic pregnancy, fibroids, IBD, IBS, etc.
Diagnostic Testing
Primary:
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Diagnosis clinical
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Tests only if refractory:
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Pregnancy test
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Urine testing, STI screen
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Pelvic US
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Secondary:
- US, MRI, laparoscopy if needed
Treatment
General Measures
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Exercise, topical heat [A,C]
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High-frequency TENS
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Treat underlying cause (for secondary)
First-Line Medications
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NSAIDs (equal efficacy; begin 1β2 days before menses):
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Ibuprofen 400 mg q8h
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Naproxen sodium 500 mg q12h
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Celecoxib 400 mg x1, then 200 mg q12h
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Mefenamic acid 500 mg x1, then 250 mg q6h
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Hormonal contraceptives:
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COCs, levonorgestrel IUD, progestins
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Continuous dosing preferred
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Estrogen-containing COCs or progestin-only for secondary
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Contraindications
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NSAIDs/COCs:
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Platelet disorders
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Gastritis/ulcers
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Smoking + migraine w/ aura
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Vascular disease, liver/kidney dysfunction
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Second-Line
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Acetaminophen Β± caffeine
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Relaxation therapy, yoga
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Nifedipine
Procedures
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Laparoscopic nerve ablation, presacral neurectomy (resistant cases)
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Hysterectomy: last resort, after completed childbearing
CAM
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Acupuncture, acupressure
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Aromatherapy massage
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Promising but less evidence: ginger, fennel, fenugreek, valerian, vitamin K1, vaginal sildenafil, etc.
Inpatient/Admission
- Outpatient management for both primary and secondary
Ongoing Care
Follow-Up
- Monitor symptom relief with treatment
Diet
- No strong evidence for dietary changes
Patient Education
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Primary dysmenorrhea is treatable and may improve with age or childbirth
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NSAIDs, hormonal options, exercise, heat are effective
Prognosis
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Primary: improves with age/parity
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Secondary: depends on etiology
Complications
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Primary: anxiety, depression
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Secondary: infertility
Clinical Pearls
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Dysmenorrhea is a leading cause of absenteeism in women <30
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All NSAIDs are equally effective when taken 1β2 days before menses
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Hormonal contraceptives preferred in women needing both symptom relief and contraception